Aeroallergen

Source: Wikipedia, the free encyclopedia.

An aeroallergen (pronounced aer·o·al·ler·gen) is any

allergic reaction
.

Pollens

Aeroallergens include the pollens of specific seasonal plants is commonly known as "

hay fever", because it is most prevalent during haying season, from late May to the end of June in the Northern Hemisphere; but it is possible to experience hay fever throughout the year.[citation needed
]

The pollen which causes hay fever varies from person to person and from region to region; generally speaking, the tiny, hardly visible pollens of wind-pollinated plants are the predominant cause. Pollens of insect-pollinated plants are too large to remain airborne and pose no risk.

Examples of plant pollen commonly responsible for hay fever include:

The time of year at which hay fever symptoms manifest themselves varies greatly depending on the types of pollen to which an allergic reaction is produced. The pollen count, in general, is highest from mid-spring to early summer. As most pollens are produced at fixed periods in the year, a person with long-term hay fever may also be able to anticipate when the symptoms are most likely to begin and end, although this may be complicated by an allergy to dust particles.[citation needed]

Spores

Spores being ejected by fungi

In fungi, both asexual and sexual spores or sporangiospores of many fungal species are actively dispersed by forcible ejection from their reproductive structures, which travel through the air over long distances. Many fungi thereby possess specialized mechanical and physiological mechanisms as well as spore-surface structures, such as

puffballs
.

Foodstuffs

It is commonly thought that peanuts and other allergic foodstuffs may become airborne, thus triggering allergic reactions in susceptible individuals, especially children.[3][4][5]

However, one report notes:

Recently concern has been raised that peanut protein in the air will trigger a full-blown anaphylaxis since respiratory exposure can occur in the school setting as food proteins aerosolize into vapors during cooking at high temperatures, even in well-ventilated cafeterias. When airborne peanut protein exposure and reactions of children with known peanut allergies were explored, no allergic symptoms or anaphylaxis were observed when peanut allergic children were not aware of the airborne exposure. Interestingly, when aware of the exposure, symptoms of itchy eyes, sneezing, and runny nose resulted. In a research article by Perry, et al. (2004), no peanut allergen was detected in the air after subjects consumed peanut butter, shelled peanuts, and unshelled peanuts. As Dr. Michael Young notes in his 2006 book, The Peanut Allergy Answer Book, predicting who will have a life-threatening anaphylactic response to airborne allergy is very unpredictable and the likelihood of it is very, very small. ... There remains no evidence that exposure to airborne peanut protein worsens allergy or results in anaphylaxis for the majority of peanut allergic individuals. There always remains the possibility that someone who is exceptionally sensitive will experience a severe reaction, however, protecting them from all possible exposures to peanut protein is extremely difficult.

— NetWellness website[6]

Eosinophilic gastroenteritis

Eosinophilic gastroenteritis (EG) is a rare and heterogeneous condition characterized by patchy or diffuse

gastrointestinal (GI) tissue, first described by Kaijser in 1937.[7][8] Aeroallergens can cause EG.[9][10]

The

As a part of

etc.

Eosinophil recruitment into inflammatory tissue is a complex process, regulated by a number of

GM-CSF) may be behind the recruitment and activation. They have been observed immunohistochemically in diseased intestinal wall.[14]
In addition eotaxin has been shown to have an integral role in regulating the homing of eosinophils into the lamina propria of stomach and small intestine.[10] In the allergic subtype of disease, it is thought that food
allergens cross the intestinal mucosa and trigger an inflammatory response that includes mast cell degranulation and recruitment of eosinophils.[9]

EG is "managed" (treated) with

leukotriene receptor antagonist) have been proposed, centering on an allergic hypothesis, with mixed results.[9][15] An elimination diet may be successful if a limited number of food allergies are identified.[16]

See also

References

  1. PMID 17784861
    .
  2. .
  3. Food Allergy & Anaphylaxis Network, in Anaphylaxis Canada, September newsletter) found at allergysafecommunities.ca Archived December 27, 2006, at the Wayback Machine
    . (.pdf) Accessed March 19, 2009.
  4. ^ "Passengers with the condition, which can be deadly, can try to ensure a peanut-free flight. But even the best plans sometimes don't work." See "Out of the Blue: Peanut allergies are a little-known danger." Elliott Hester, St. Petersburg Times, December 30, 2001, St. Petersburg Times. Accessed March 19, 2009.
  5. ^ Constance Hays, "Ideas & Trends: Airborne Allergies; A New Fear of Flying: Peanuts", The New York Times, Sunday, May 10, 1998, found at NY Times archives. Accessed March 19, 2009.
  6. ^ Jill F. Kilanowski & Ann Stalter (College of Nursing, The Ohio State University), "Children's Health: Peanut Allergy in the School Environment: Myths and Facts: Part 1 of a 2-Part Series", at NetWellness website Archived February 27, 2009, at the Wayback Machine, citing Perry, T., Conover-Walker, M., Pomes, A., Chapman, M., & Wood, R. (2004). "Distribution of peanut allergen in the environment." Journal of Allergy and Clinical Immunology, 113, 973-976. Accessed March 19, 2009.
  7. ^ Kaijser R. Zur Kenntnis der allergischen Affektionen des Verdauugskanals vom Standpunkt des Chirurgen aus. Arch Klin Chir 1937; 188:36–64.
  8. PMID 15028974
    .
  9. ^ .
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