Oscillopsia
Oscillopsia is a
nystagmus, or impaired coordination in the visual cortex (especially due to toxins) and is one of the symptoms of superior canal dehiscence syndrome. Those affected may experience dizziness and nausea. Oscillopsia can also be used as a quantitative test to document aminoglycoside toxicity. Permanent oscillopsia can arise from an impairment of the ocular system that serves to maintain ocular stability.[2] Paroxysmal oscillopsia can be due to an abnormal hyperactivity in the peripheral ocular or vestibular system.[2]
Symptoms
Patients may feel wobbly vision, back and forth vibrating, blurred vision, and different symptoms depending on the severity of the problem.
During a visual symptom, patients may become dizzy, or nauseous. Closing your eyes during this may not always work, as you will still have feeling of eye movement. While it may not happen, the dizziness effect could cause anyone to throw-up, but this does not always happen.
Permanent oscillopsia due to impairment of ocular stabilizing systems
Ocular stability is maintained by three different ocular motor systems
1. The fixation system and its deficit
- In the fixation system, the ocular motor noise that comes from microsaccades, microtremors and slow drifts (all necessary for important perceptual functions) are limited by the visual and
- A deficit in this fixation system results in ocular instability that mainly leads to acquired pendular nystagmus and saccadic intrusions. Acquired pendular nystagmus is seen in a variety of conditions with the two most frequent being multiple sclerosis and oculopalatal tremor.[2]
2. The visuo-vestibular stabilizing systems and their deficits
- The vestibular and visual ocular stabilizing systems interact together in order to maintain the image of the visual scene steady on the retina during a head and body displacement situation.[2]
- A deficit in these vestibular or visual ocular stabilizing systems may result in ocular instability due to pathological jerk vestibulo-ocular reflex deficit (especially when bilateral) and a deficit of vestibulo-ocular reflex inhibition can result in oscillopsia and impaired visual acuity during head and body displacement.[2]
3. The neural integrator and its deficit
References
- ^ Gold, Daniel. "Oscillopsia: a common symptom of bilateral vestibular loss". Neuro-Ophthalmology Virtual Education Library(NOVEL, NOVEL.utah.edu): Daniel Gold Collection. Spencer S. Eccles Health Sciences Library. Retrieved 2019-11-20.
- ^ PMID 21102332.
- PMID 14748767. Retrieved 6 May 2012.