Precordial catch syndrome

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Precordial catch syndrome
Other namesTexidor's twinge[1]
One of the areas more commonly affected in precordial catch syndrome[1]
SpecialtyPediatrics, family medicine
SymptomsSharp, stabbing chest pain in a small area[1]
Usual onsetSudden[1]
Duration30 seconds to 3 minutes[1]
CausesUnclear[1]
Differential diagnosisAngina, pericarditis, pleurisy, chest trauma[1]
TreatmentReassurance[1]
PrognosisGood[1]
FrequencyRelatively common[1]

Precordial catch syndrome (PCS) is a non-serious condition in which there are sharp stabbing pains in the chest. These typically get worse with inhaling and occur within a small area. Spells of pain usually last less than a few minutes. Typically it begins at rest and other symptoms are absent. Concerns about the condition may result in anxiety.[1]

The underlying cause is unclear. Some believe the pain may be from the chest wall or irritation of an

intercostal nerve.[1][2] Risk factors include psychological stress.[2] The pain is not due to the heart. Diagnosis is based on the symptoms. Other conditions that may produce similar symptoms include angina, pericarditis, pleurisy, and chest trauma.[1]

Treatment is usually via reassurance, as the pain generally resolves without any specific treatment. Precordial catch syndrome is relatively common, and children between the ages of 6 and 12 are most commonly affected. Males and females are affected equally.[1] It is less common in adults.[2] The condition has been described since at least 1893.[3]

Signs and symptoms

Characteristic symptoms include sharp stabbing pains in the chest. These typically get worse with breathing in and occur within a small area. Spells of pain usually last less than a few minutes. Typically it begins at rest and other symptoms are absent. Concerns about the condition may result in anxiety, with sufferers often fearing that the pain is a sign of a more serious condition. Similar anxieties in those who experience the syndrome on a regular basis may manifest as a worry of the syndrome itself happening, with patients feeling scared to take fuller breaths in fear of triggering a spell.[1]

Causes

The underlying cause is unclear.

intercostal nerve.[1][2] Psychological stress is correlated with precordial catch syndrome.[2] The pain is not due to the heart.[1]

Treatment

Treatment is usually via reassurance, as the pain generally resolves without any specific treatment. Occasionally it goes away after a couple of breaths.[1]

There is no known cure for PCS; however, PCS is not believed to be dangerous or life-threatening. Many see the worst part about PCS to be the fear that this chest pain is an indicator of a

heart attack
or other more serious condition. As the condition is not dangerous or life-threatening, there is no reason to take medication, although some people may choose to refrain from some normal activities such as physical exercise, as this can exacerbate the pain, particularly if it occurs during physical activity.

History

The syndrome was first described and named in 1893 by

cardiologist, who called it "précordialgie" (from the latin "praecordia" meaning "before the heart"), or "Syndrôme de Huchard" ("Huchard syndrome").[4][5] The term "precordial" had entered the French medical lexicon with the 1370 translation of Guy de Chauliac's Chirurgia magna.[6] Previously, the Latin term "praecordia" had been used to refer to the diaphragm, a sense now obsolete.[6][7]

The Huchard syndrome was then studied more deeply by Miller and Texidor, medical practitioners at the Cardiovascular Department and the Department of Medicine at the Michael Reese Hospital in Chicago, in 1955.[8] They reported the condition in 10 patients, one being Miller himself. In 1978, PCS was discussed by Sparrow and Bird who reported 45 with it, and that it was probably more frequent than generally assumed.[9] PCS in American children has been discussed by Pickering in 1981[10] and by Reynolds in 1989.[11] Gumbiner reviews PCS as a diagnosis in his 2003 article.[1] Incidences of PCS in swimmers with asthma was analyzed in Hayes, et al.'s article in 2016, constituting the most recent English-language article on the syndrome as of May 2023.[12]

See also

References

External links