Tachycardia
Tachycardia | |
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Other names | Tachyarrhythmia |
ECG showing sinus tachycardia with a rate of about 100 beats per minute | |
Pronunciation |
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Specialty | Cardiology |
Differential diagnosis |
Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate.[1] In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults.[1] Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart).
Complications
Tachycardia can lead to
When the rate of blood flow becomes too rapid, or fast blood flow passes on damaged endothelium, it increases the friction within vessels resulting in turbulence and other disturbances.[3] According to the Virchow's triad, this is one of the three conditions that can lead to thrombosis (i.e., blood clots within vessels).[4]
Causes
Some causes of tachycardia include:[5]
- Adrenergic storm
- Anaemia
- Anxiety
- Atrial fibrillation
- Atrial flutter
- Atrial tachycardia
- Atrioventricular reentrant tachycardia
- AV nodal reentrant tachycardia
- Brugada syndrome
- Circulatory shock and its various causes (obstructive shock, cardiogenic shock, hypovolemic shock, distributive shock)
- Dehydration
- Dysautonomia
- Exercise
- Fear
- Hypoglycemia
- Hypovolemia
- Hyperthyroidism
- Hyperventilation
- Inappropriate sinus tachycardia
- Junctional tachycardia
- Metabolic myopathy
- Multifocal atrial tachycardia
- Pacemakermediated
- Pain
- Panic attack
- Pheochromocytoma
- Sinus tachycardia
- Sleep deprivation[6]
- Supraventricular tachycardia
- Ventricular tachycardia
- Wolff–Parkinson–White syndrome
Drug related:
- Alcohol (Ethanol) intoxication
- Stimulants
- Cannabis
- Drug withdrawal
- Tricyclic antidepressants
- Nefopam
- Opioids (rare)
Diagnosis
The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below:[7][8]
- 1–2 days: Tachycardia >159 beats per minute (bpm)
- 3–6 days: Tachycardia >166 bpm
- 1–3 weeks: Tachycardia >182 bpm
- 1–2 months: Tachycardia >179 bpm
- 3–5 months: Tachycardia >186 bpm
- 6–11 months: Tachycardia >169 bpm
- 1–2 years: Tachycardia >151 bpm
- 3–4 years: Tachycardia >137 bpm
- 5–7 years: Tachycardia >133 bpm
- 8–11 years: Tachycardia >130 bpm
- 12–15 years: Tachycardia >119 bpm
- >15 years – adult: Tachycardia >100 bpm
Heart rate is considered in the context of the prevailing clinical picture. When the heart beats excessively or rapidly, the heart pumps less efficiently and provides less blood flow to the rest of the body, including the heart itself. The increased heart rate also leads to increased work and oxygen demand by the heart, which can lead to rate related ischemia.[9]
Differential diagnosis
An
- Narrow complex
- sino-atrial (SA) node, near the base of the superior vena cava
- Atrial fibrillation
- Atrial flutter
- AV nodal reentrant tachycardia
- Accessory pathway mediated tachycardia
- Atrial tachycardia
- Multifocal atrial tachycardia
- Cardiac Tamponade
- Junctional tachycardia (rare in adults)
- Wide complex
- Ventricular tachycardia, any tachycardia that originates in the ventricles
- Any narrow complex tachycardia combined with a problem with the conduction system of the heart, often termed "supraventricular tachycardia with aberrancy"
- A narrow complex tachycardia with an accessory conduction pathway, often termed "supraventricular tachycardia with pre-excitation" (e.g. Wolff–Parkinson–White syndrome)
- Pacemaker-tracked or pacemaker-mediated tachycardia
Tachycardias may be classified as either narrow complex tachycardias (supraventricular tachycardias) or wide complex tachycardias. Narrow and wide refer to the width of the
Sinus
The body has several
Upon exertion, sinus tachycardia can also be seen in some inborn errors of metabolism that result in metabolic myopathies, such as McArdle's disease (GSD-V).[12][13] Metabolic myopathies interfere with the muscle's ability to create energy. This energy shortage in muscle cells causes an inappropriate rapid heart rate in response to exercise. The heart tries to compensate for the energy shortage by increasing heart rate to maximize delivery of oxygen and other blood borne fuels to the muscle cells.[12]
"In McArdle's, our heart rate tends to increase in what is called an 'inappropriate' response. That is, after the start of exercise it increases much more quickly than would be expected in someone unaffected by McArdle's."[14] As skeletal muscle relies predominantly on glycogenolysis for the first few minutes as it transitions from rest to activity, as well as throughout high-intensity aerobic activity and all anaerobic activity, individuals with GSD-V experience during exercise: sinus tachycardia, tachypnea, muscle fatigue and pain, during the aforementioned activities and time frames.[12][13] Those with GSD-V also experience "second wind", after approximately 6–10 minutes of light-moderate aerobic activity, such as walking without an incline, where the heart rate drops and symptoms of exercise intolerance improve.[12][13][14]
An increase in
Inappropriate sinus tachycardia
Inappropriate sinus tachycardia (IST) is a diagnosis of exclusion,[16] a rare but benign type of cardiac arrhythmia that may be caused by a structural abnormality in the sinus node. It can occur in seemingly healthy individuals with no history of cardiovascular disease. Other causes may include autonomic nervous system deficits, autoimmune response, or drug interactions. Although symptoms might be distressing, treatment is not generally needed.[17]
Ventricular
Ventricular tachycardia (VT or V-tach) is a potentially life-threatening cardiac arrhythmia that originates in the ventricles. It is usually a regular, wide complex tachycardia with a rate between 120 and 250 beats per minute. A medically significant subvariant of ventricular tachycardia is called torsades de pointes (literally meaning "twisting of the points", due to its appearance on an EKG), which tends to result from a long QT interval.[18]
Both of these rhythms normally last for only a few seconds to minutes (paroxysmal tachycardia), but if VT persists it is extremely dangerous, often leading to ventricular fibrillation.[19][20]
Supraventricular
This is a type of tachycardia that originates from above the ventricles, such as the atria. It is sometimes known as paroxysmal atrial tachycardia (PAT). Several types of supraventricular tachycardia are known to exist.[21]
Atrial fibrillation
AV nodal reentrant tachycardia
AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a regular narrow complex tachycardia that usually responds well to the Valsalva maneuver or the drug adenosine. However, unstable patients sometimes require synchronized cardioversion. Definitive care may include catheter ablation.[23]
AV reentrant tachycardia
Junctional tachycardia
Junctional tachycardia is an automatic tachycardia originating in the AV junction. It tends to be a regular, narrow complex tachycardia and may be a sign of digitalis toxicity.[24]
Management
The management of tachycardia depends on its type (wide complex versus narrow complex), whether or not the person is stable or unstable, and whether the instability is due to the tachycardia.[10] Unstable means that either important organ functions are affected or cardiac arrest is about to occur.[10]
Unstable
In those that are unstable with a narrow complex tachycardia, intravenous adenosine may be attempted.[10] In all others immediate cardioversion is recommended.[10]
Terminology
The word tachycardia came to English from
See also
References
- ^ ISBN 9781405104647.
- ^ Thompson EG, Pai RK, eds. (2 June 2011). "Passing Out (Syncope) Caused by Arrhythmias". CardioSmart. American College of Cardiology. Archived from the original on 2020-06-13. Retrieved 2020-04-13.
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- ^ "Supraventricular Tachycardias". The Lecturio Medical Concept Library. 9 September 2020. Retrieved 2 July 2021.
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- ISBN 978-0-323-07688-3.
- ^ Kantharia BK, Sharma M, Shah AN (17 October 2021). "Atrial Tachycardia: Practice Essentials, Background, Anatomy". MedScape. WebMD LLC.
- ISBN 978-0-07-146633-2.
- ^ PMID 20956224.
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- ^ PMID 34848128.
- ^ PMID 25054987.
- ^ a b Wakelin A (2017). Living With McArdle Disease (PDF). International Assoc. of Muscle Glycogen Diseases (IAMGSD). p. 15.
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- PMID 35710196.
- S2CID 23249973.
- ^ Mitchell LB (January 2023). "Torsades de Pointes Ventricular Tachycardia". Merck Manual Profesional Edition. Retrieved 19 April 2019.
- PMID 11334828.
- S2CID 207215117.
- ^ "Types of Arrhythmia". National Heart, Lung, and Blood Institute (NHLBI). U.S. National Institutes of Health. July 1, 2011. Archived from the original on June 7, 2015.
- ^ Oiseth S, Jones L, Maza E (11 August 2020). "Atrial Fibrillation". The Lecturio Medical Concept Library. Retrieved 3 July 2021.
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- ^ Elsevier, Dorland's Illustrated Medical Dictionary, Elsevier.
- ^ Merriam-Webster, Merriam-Webster's Medical Dictionary, Merriam-Webster.
- ^ a b Wolters Kluwer, Stedman's Medical Dictionary, Wolters Kluwer.
- ^ Houghton Mifflin Harcourt, The American Heritage Dictionary of the English Language, Houghton Mifflin Harcourt.
- ^ Merriam-Webster, Merriam-Webster's Collegiate Dictionary, Merriam-Webster, archived from the original on 2020-10-10, retrieved 2017-07-22.
- ^ Merriam-Webster, Merriam-Webster's Unabridged Dictionary, Merriam-Webster, archived from the original on 2020-05-25, retrieved 2017-07-22.