Action potential pulse
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An action potential pulse is a mathematically and experimentally correct Synchronized Oscillating Lipid Pulse coupled with an Action Potential. This is a continuation of Hodgkin Huxley's work in 1952 with the inclusion of accurately modelling ion channel proteins, including their dynamics and speed of activation. [1][2][3]
The action potential pulse is a model of the speed an action potential that is dynamically dependent upon the position and number of ion channels, and the shape and make up of the axon. The action potential pulse model takes into account entropy and the conduction speed of the action potential along an axon. It is an addition to the Hodgkin Huxley model.
Investigation into the
The resulting action potential pulse therefore is a synchronized, coupled pulse with the entropy from depolarization at one channel providing sufficient entropy for a pulse to travel to sequential channels and mechanically open them.
This mechanism explains the speed of transmission through both myelinated and unmyelinated axons.
This is a timed pulse, that combines the entropy from ion transport with the efficiency of a flowing pulse.
The action potential pulse model has many advantages over the simpler Hodgkin Huxley version including evidence, efficiency, timing entropy measurements, and the explanation of nerve impulse flow through myelinated axons.
Myelinated axons
This model replaces saltatory conduction, which was a historical theory that relied upon cable theory to explain conduction, and was an attempt at a model that has no basis is either physiology or membrane biophysics.
In myelinated axons the myelin acts as a mechanical transducer preserving the entropy of the pulse and insulating against mechanical loss. In this model the