Rasmussen aneurysm
It is caused by the progressive thinning of the pulmonary artery wall. This weakening process is characterized by the replacement of both the tunica externa and tunica media with granulation tissue, which subsequently undergoes transformation into fibrin. This tissue remodeling is a consequence of the healing response in the associated lung cavity. The dilation of the pulmonary artery in close proximity to or involvement within the lung cavity leads to the formation of a pseudoaneurysm.[2][4] As is typical with any aneurysm, Rasmussen aneurysm carries the inherent risk of rupture, which may result in life-threatening massive hemoptysis, characterized by the coughing of blood. Such events are associated with a mortality rate exceeding 50%.[2][5]
Historically, Rasmussen aneurysms were widely regarded as a common etiology of hemoptysis, particularly in tuberculosis cases. However, with advancements in
Rasmussen aneurysm was initially associated exclusively with cavitary tuberculosis, but the term is now utilized to encompass any anatomical aneurysm occurring in conjunction with various forms of destructive lung lesions.[medical citation needed]
References
- , retrieved 2023-09-15
- ^ PMID 34235447.
- ^ Rasmussen, V. On hemoptysis, especially when fatal, in its anatomical and clinical aspects. (Translated from the Hospitals-Tidende, 11th year, nos. 9–13, Copenhagen, February and March, 1868, by Moore WD). Edinburgh Med J. 1868; 14: 385–401
- ^ PMID 29410220.
- S2CID 46627528.
- PMID 3053058.
- PMID 26744661.