Accelerans nerve

Source: Wikipedia, the free encyclopedia.

Accelerans nerve forms a part of the sympathetic branch of the

blood flow, preparing the body for a sudden increase in activity.[3][4][5] These nerve fibres are part of the autonomic nervous system
, part of the 'fight or flight' system.

Right where the sinus-atrial node is, the negative charge of the interior of the fibres of heart muscles breaks down spontaneously the cells in the pacemaker about 70 minutes each time.[6] As a result of this, a small current sweeps over the atria, which then reaches the insulating connective tissue between the atria and ventricles. When the current reaches this part, the atrio-ventricular node picks it up, thus creating the rhythm of the heartbeats through the rate and strength of each palpitation which is controlled by the accelerans nerve.[2][4]

History and etymology

The accelerans nerve was first discovered on April 23, 1883 by German medical students L.C. Wooldridge, D.S and George Henry Lewes, M.B.

noradrenaline
back to the accelerans nerve.

The term accelerans comes from the latin word accelerrō which means "quickening" or "to hasten". The term

sinew or tendon
in an animal body.

Measurements in mammals

The majority of research regarding the accelerans nerve has been centered around other mammals part from humans. The first study ever conducted on accelerans nerve on April 23, 1883 necessitated other mammals apart from humans, not only due to ethical concerns, but in order to visualize the difference in their stimulation depending on the mammal.[5] The following is a list of mammals who have been used for research into the accelerans nerve and what results were found from the studies:

Dogs

In the case of dogs, the first case ever mentioned regarding the accelerans nerve and its function in 1883 yielded that the investigation in dogs was done almost exclusively on the right side of the nerves.

noradrenaline.[9] These results indicated that in medical situations in which the rate and strength of a patient's heartbeats reaches critically low levels, stimulation of the accelerans nerve allows for a more expeditious way to gain the heart's vitality back, analogous to the function of artificial pacemakers
.

Cats

In the case of cats, stimulation of the accelerans nerve was found to produce a much more significant effect on their heart rates compared to the hormones secreted by the adrenals glands in the autonomic-nervous system for the fight-or-flight response.[10] Moreover, the vigorous heart palpitations induced by the accelerans nerve in the hearts of cats seem to disappear quickly after a low-frequency stimulation is applied to the nerves.[2][5][10] This quick shift in the heart rate resulted in the idea that decreases of stroke volume in humans requires direct vagal control of the heart ventricles.

Rabbit

In the case of rabbits, a study conducted in December 1973 directly administered certain substances to slow down the heart rate ranging from

noradrenaline due to the stimulation of the accelerans nerve by 5 hertz. In another case, it was observed that acidic substances, specifically acetic acid, when exposed to the accelerans nerve in rabbit hearts led to an increase in blood pressure
due to the nerve's reaction with the acid which made it send out stronger signals.

See also

References

  1. ISSN 1040-1237
    .
  2. ^
    ISBN 978-0-511-80182-2
    .
  3. ^ "The Open Door Web Site : Major Factors Controlling the Heartbeat". Archived from the original on 29 December 2018. Retrieved 25 October 2019.
  4. ^ a b In Search of Memory: The Emergence of a New Science of Mind - Eric R. Kandel Google Books
  5. ^
    JSTOR 114374
    .
  6. ^ a b "15.3C: The Heartbeat". Biology LibreTexts. 2016-07-13. Retrieved 2023-04-21.
  7. PMID 6273755
    – via Macmillan Journals Ltd.
  8. PMID 18137898
    – via PubMed.
  9. PMID 2905183
    – via University of Cagliari.
  10. ^
    PMID 13372369
    – via Wiley Online Library.
  11. PMID 4788036
    .
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